T1R2 and T1R3 knockouts demonstrated significant attenuation of fructose-induced, salt-sensitive hypertension

dc.contributor.authorEaton, Andrew J.
dc.date.accessioned2022-04-06T14:21:39Z
dc.date.available2022-04-06T14:21:39Z
dc.date.issued2018
dc.description1 Broadside. 48"W x 36"Hen_US
dc.description.abstractIncreased fructose consumption apparent in the Western diet leads to an increased risk for hypertension, or chronic high blood pressure. In cases of individuals pre-disposed to volatile blood pressure increase in the presence of a high-salt diet, salt-sensitive hypertension can develop due to excess fructose absorption in the gastrointestinal tract. Taste receptors on the tongue, namely of the T1R family, play a key role in fructose absorption. Specific receptors T1R2 and T1R3 transmit nerve signals to the brain, initiating absorption of fructose in the gastrointestinal tract. The reliance on T1Rs to control fructose absorption makes them an intriguing cellular component to attempt to moderate hypertension onset.en_US
dc.description.sponsorshipKalamazoo College. Department of Biology. Diebold Symposium, 2018en_US
dc.format.mimetypeapplication/pdf
dc.identifier.urihttps://cache.kzoo.edu/handle/10920/43366
dc.language.isoen_USen_US
dc.publisherKalamazoo, Mich. : Kalamazoo Collegeen_US
dc.relation.ispartofKalamazoo College Diebold Symposium Presentation Collectionen
dc.rightsU.S. copyright laws protect this material. Commercial use or distribution of this material is not permitted without prior written permission of the copyright holder.en
dc.titleT1R2 and T1R3 knockouts demonstrated significant attenuation of fructose-induced, salt-sensitive hypertensionen_US
dc.typePresentationen_US
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