dc.contributor.author | Teasley, Hanna E. | |
dc.date.accessioned | 2022-04-08T13:50:56Z | |
dc.date.available | 2022-04-08T13:50:56Z | |
dc.date.issued | 2018 | |
dc.identifier.uri | https://cache.kzoo.edu/handle/10920/43392 | |
dc.description | 1 Broadside. 48"W x 36"H | en_US |
dc.description.abstract | Steroid hormones are essential regulators of uterine function.
AT-rich interaction domain 1A protein (ARID1A) plays an
important role in steroid hormone signaling in endometrial
function and pregnancy. Phosphatidylinositol-3-kinase
interacting protein 1 (Pik3ip1), a known negative regulator of
PI3K induced cell division and survival, is a direct
downstream target of ARID1A, making it a gene of interest
within endometriosis and female fertility. | en_US |
dc.description.sponsorship | Kalamazoo College. Department of Biology. Diebold Symposium, 2018 | en_US |
dc.format.mimetype | application/pdf | |
dc.language.iso | en_US | en_US |
dc.publisher | Kalamazoo, Mich. : Kalamazoo College | en_US |
dc.relation.ispartof | Kalamazoo College Diebold Symposium Presentation Collection | en |
dc.rights | U.S. copyright laws protect this material. Commercial use or distribution of this material is not permitted without prior written permission of the copyright holder. | en |
dc.title | Steroid hormone regulation of PIK3IP1, a known suppressor of PI3K induced cell division and survival, in the mouse uterus | en_US |
dc.type | Presentation | en_US |