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dc.contributor.advisorMiller, William F.
dc.contributor.authorCarlson, Lynne
dc.contributor.otherCarlson, Evelyn Ethel
dc.date.accessioned2010-07-06T14:24:47Z
dc.date.available2010-07-06T14:24:47Z
dc.date.issued1969
dc.identifier.urihttp://hdl.handle.net/10920/15965
dc.descriptionv, 49 p.en_US
dc.description.abstractData is presented to substantiate the hypothesis that serum alpha 1- antitrypsin deficiency is sometimes a factor in the occurrence of pulmonary emphysema. This theory is particularly valid when emphysema is manifest in patients under 40 years of age. The proposed role of alpha 1 -antitrypsin is to destroy the proteolytic enzymes released by leukocytes in the process of fighting infection. Diminished amounts of alpha 1 - antitrypsin are not sufficient to protect the lungs from the inflammatory exudate; this results in destruction of lung tissue. It is further postulated that alpha 1 -antitrypsin deficiency is transmitted by an autosomal recessive gene, a theory borne out by the pedigrees of the families studied.en_US
dc.description.sponsorshipCardiopulmonary Institute. Southwestern Medical School and Methodist Hospital. University of Texas, Dallas, Texas.
dc.format.mimetypeapplication/pdf
dc.language.isoen_USen_US
dc.publisherKalamazoo Collegeen_US
dc.relation.ispartofKalamazoo College Biology Senior Individualized Projects Collection
dc.rightsU.S. copyright laws protect this material. Commercial use or distribution of this material is not permitted without prior written permission of the copyright holder.
dc.titleAlpha1-Antitrypsin Deficiency and Pulmonary Deficiencyen_US
dc.typeThesisen_US


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  • Biology Senior Individualized Projects [1489]
    This collection includes Senior Individualized Projects (SIP's) completed in the Biology Department. Abstracts are generally available to the public, but PDF files are available only to current Kalamazoo College students, faculty, and staff.

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